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Transcriptome analysis of microglia isolated from SOD1 G93A mice revealed increases in activated pro-inflammatory Igf1 , Progranulin, Trem2 , cytokines, and neurotoxic factor MMP-12 (Chiu et al., G93A and SOD1 L8Q stimulate Rac1-GTP activation of NOX and the production of ROS, unlike WT SOD1 (Boille et al., M1 microglial cells are also activated by inflammatory stimuli

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Synchronization and rhythmic processes in physiology

ROS provides several wound protection mechanisms by decreasing blood flow and activating cellular signals responsible for thrombus formation

Utilizing relevant animal models that closely mimic human disease conditions can provide critical insights into the safety and efficacy of new treatments